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Ation and glial cytoplasmic inclusions. Ann Neurol 1996, 39(2):241-255. 49. Burk K, Globas C, Wahl T, Buhring U, Dietz K, Zuhlke C, Luft A, Schulz JB, Voigt K, Dichgans J: MRI-based volumetric differentiation of sporadic cerebellar ataxia. Brain 2004, 127(Pt 1):175-181.50. Dickson DW, Lin W, Liu WK, Yen SH: Multiple system atrophy: a sporadic synucleinopathy. Brain Pathol 1999, 9(4):721-732. 51. J
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Ed in participating in a randomised trial, of which 4295 women were found to be eligible according to the preliminary assessment. The eligibility criteria included age of 50 to 64 and an elapsed time of 12 months or more since the last period at the randomisation stage [21]. A personal invitation was mailed to eligible women in 1999?001. A total of 2323 women responded to the invitation to visit t
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Ving roles in a variety of biological processes that are relevant to our experimental model. These include infection by bacteria, inflammatory response, complement activation, phagocytosis of macrophages, and others. Proteins that wereTable 4: List of proteins with different changes for strains of mice between 4 hours and 24 hours post infectionGel No. 3 12 13 14 16 25 26 29 31 38 40 42 45 49 50 5
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Ki M, Kitagaki H, Yamaji S, Sakamoto S, Matsuda K, Mori E: Reduction of cerebellar glucose metabolism in advanced Alzheimer's disease. J Nucl Med 1997, 38(6):925-928. 58. Larner AJ: The cerebellum in Alzheimer's disease. Dement Geriatr Cogn Disord 1997, 8(4):203-209. 59. Wegiel J, Wisniewski HM, Dziewiatkowski J, Badmajew E, Tarnawski M, Reisberg B, Mlodzik B, De Leon MJ, Miller DC: Cerebellar atr
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